Dr. Inês M. Araújo

Assistant Professor (UAlg) and Group Leader (CBME)
University of Algarve (UAlg)
Department of Biomedical Sciences and Medicine
and Centre for Molecular and Structural Biomedicine (CBME)
Gambelas Campus
Edifício 7, 3º Piso
8005-139 Faro, Portugal

T: +351 289 800094, ext 7534
F: +351 289 800076
E: This email address is being protected from spambots. You need JavaScript enabled to view it.

Research Interests

  • injury induced-neurogenesis
  • modification of adult neurogenesis by nitric oxide
  • physiological role of calpains in neurogenesis
  • prenatal exposure to drugs and brain development
  • neurogenesis and inflammation

Selected Recent Publications

  •  Martinez-Ruiz A, Araújo IM, Izquierdo-Álvarez A, Hernansanz-Agustín P, Lamas S, Serrador JM (2013) Specificity in S-nitrosylation: a short-range mechanism for NO signaling? Antioxidants and Redox Signaling, Epub Jan 4, 2013
  • Carreira BP, Morte MI, Inácio Â, Rosmaninho-Salgado J, Costa G, Agasse F, Carmo A, Couceiro P, Brundin P, Ambrósio AF, Carvalho CM, Araújo IM (2010) Nitric oxide stimulates the proliferation of neural stem cells bypassing the epidermal growth factor receptor. Stem Cells 28(7):1219-1230.
  • Araújo IM, Gil J, Carreira BP, Mohapel P, Petersén Å, Pinheiro PS, Soulet D, Bahr BA, Brundin P, Carvalho CM (2008) Calpain activation is involved in early caspase-independent neurodegeneration in the hippocampus following status epilepticus. Journal of Neurochemistry 105(3):666-676
  • Yu L, Shen H-Y, Coelho JE, Araújo IM, Huang Q-Y, Day Y-J, Rebola N, Canas PM, Rapp EK, Ferrara J, Taylor D, Müller CE, Linden J, Cunha RA, Chen J-F (2008) A2A receptors modulate motor activity and MPTP neurotoxicity by distinct cellular mechanisms. Annals of Neurology 63(3):338-34
  • Araújo IM*, Carreira BP*, Pereira T, Santos PF, Soulet D, Inácio Â, Bahr BA, Carvalho AP, Ambrósio AF, Carvalho CM (2007) Changes in calcium dynamics following the reversal of the sodium-calcium exchanger have a key role in AMPA receptor-mediated neurodegeneration via calpain activation in hippocampal neurons. Cell Death and Differentiation 14(9):1635-1646.



POLICY STATEMENT: Biomedicine and Molecular Biosciences Action BM1005 is funded by COST, through its implementing agent the European Science Foundation.